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Peer Review
All the articles listed here were peer reviewed before they were published. Peer review is the process
of subjecting an author's research to the scrutiny of others who are experts in the
same field. It is difficult for authors and researchers, whether individually or in
a team, to spot every mistake or flaw in a complicated piece of work. This is not necessarily
a reflection on those concerned, but because with a new and perhaps eclectic subject, an
opportunity for improvement may be more obvious to someone with special expertise or who
simply looks at it with a fresh eye. Therefore, showing work to others increases the
probability that weaknesses will be identified and improved. For both grant-funding and
publication in a scholarly journal, it is also normally a requirement that the subject
is both novel and substantial. Peer review is generally considered essential to academic
quality.
Impact Fator
The Impact factor was devised by Eugene Garfield, the founder of the
Institute for Scientific Information, now part of Thomson, a large worldwide
US-based publisher. Impact factors are calculated each year by the Institute for
Scientific Information for those journals which it indexes, and the factors and
indices are published in Journal Citation Reports.
Though the impact factor was originally intended as an objective measure of the
reputability of a journal (Garfield), it is now being increasingly applied to
measure the productivity of scientists. The way it is customarily used is to
examine the impact factors of the journals in which the scientist's articles
have been published. This has obvious appeal for an academic administrator who
knows neither the subject nor the journals.
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Prediction of BRCA status
CML - Breast Cancer | 2011 Jul | 23(2):41-50
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Specific genomic aberrations in primary colorectal cancer are associated with liver metastases.
BMC Cancer | 2010 Dec 2 | 10(1):662
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Genomic signature of BRCA1 deficiency in sporadic basal-like breast tumors.
Genes Chromosomes Cancer | 2010 Nov 22 | [Epub ahead of print]
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Cross-species comparison of aCGH data from mouse and human BRCA1- and BRCA2-mutated breast cancers.
BMC Cancer | 2010 Aug 24 | 10(1):455
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Prediction of BRCA2-association in hereditary breast carcinomas using array-CGH.
Breast Cancer Research and Treatment | 2010 Jul 8 | [Epub ahead of print]
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Integration of DNA Copy Number Alterations and Prognostic Gene Expression Signatures in Breast Cancer Patients.
Clinical Cancer Research | 2010 Jan 12 | 16(2): 651-63
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BRCA1-deficient mammary tumor cells are dependent on EZH2 expression and sensitive to Polycomb Repressive Complex 2-inhibitor 3-deazaneplanocin A.
Breast Cancer Research | 2009 Aug 26 | 11(4):R63
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Deletion of Exons 1a-2 of BRCA1: A Rather Frequent Pathogenic Abnormality.
Genetic Testing and Molecular Biomarkers | 2009 Apr 30 | 13(3): 399-406
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Array comparative genomic hybridization identifies a distinct DNA copy number profile in renal cell cancer associated with hereditary leiomyomatosis and renal cell cancer.
Genes, Chromosomes, and Cancer | 2009 Apr 16 | 48:544–551
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High incidence of protein-truncating TP53 mutations in BRCA1-related breast cancer.
Cancer Research | 2009 Apr 15 | 69(8):3625-33
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Prediction of BRCA1-association in hereditary non-BRCA1/2 breast carcinomas with array-CGH.
Breast Cancer Research and Treatment | 2008 Aug 14 | 116(3):479-89
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ESR1 gene amplification in breast cancer: a common phenomenon?
Nature Genetics | 2008 Jul | 40(7):807-8
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Pathogenicity of the BRCA1 missense variant M1775K is determined by the disruption of the BRCT phosphopeptide-binding pocket: a multi-modal approach.
European Journal of Human Genetics | 2008 Jul | 16(7):820-32
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Automated array-CGH optimized for archival formalin-fixed, paraffin-embedded tumor material.
BMC Cancer | 2007 Mar 7 | 7:43
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A multiplex PCR predictor for aCGH success of FFPE samples.
British Journal of Cancer | 2006 Jan 30 | 94(2):333-7
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SIRAC: Supervised Identification of Regions of Aberration in aCGH datasets.
BMC Bioinformatics | 2007 Oct 30 | 8:422
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Analysis of PALB2/FANCN-associated breast cancer families.
Proceedings of the National Academy of Sciences of the United States of America (PNAS) | 2007 Apr 17 | 104(16):6788-93
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Reactive oxygen species play no role in the candidacidal activity of the salivary antimicrobial peptide histatin 5.
Biochemical Journal | 2004 Jul 15 | 381(Pt 2):447-52
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